Guidelines | Muscle/Exercise | Neurological | Vascular | Immunological | Metabolic | Coagulation | Amyloid | Patient Safety
A Long COVID Definition: A Chronic, Systemic Disease State with Profound Consequences
Myalgic encephalomyelitis or encephalopathy/chronic fatigue syndrome: diagnosis and management
Beyond Myalgic Encephalomyelitis/Chronic Fatigue Syndrome: Redefining an Illness
Long COVID: major findings, mechanisms and recommendations
Long Covid in Aotearoa NZ: Risk assessment and preventive action urgently needed
Characterizing Long COVID in Children and Adolescents
NICE guideline on ME/CFS: robust advice based on a thorough review of the evidence
The metabolic and physiologic impairments underlying long COVID associated exercise intolerance
Muscle abnormalities worsen after post-exertional malaise in long COVID
Dysregulation of extracellular vesicle protein cargo in female myalgic encephalomyelitis/chronic fatigue syndrome cases and sedentary controls in response to maximal exercise
Differential Cardiopulmonary Hemodynamic Phenotypes in PASC Related Exercise Intolerance
Post-COVID exercise intolerance is associated with capillary alterations and immune dysregulations in skeletal muscles
Unexplained exertional intolerance associated with impaired systemic oxygen extraction
Inability of myalgic encephalomyelitis/chronic fatigue syndrome patients to reproduce VO2peak indicates functional impairment
Blood-brain barrier disruption and sustained systemic inflammation in individuals with long COVID-associated cognitive impairment
Worsening Symptoms Is Associated with Larger Cerebral Blood Flow Abnormalities during Tilt-Testing in Myalgic Encephalomyelitis/Chronic Fatigue Syndrome ME/CFS
Long COVID Is Not a Functional Neurologic Disorder
Reduced Stroke Volume and Brain Perfusion Drive Postural Hyperventilation in Postural Orthostatic Tachycardia Syndrome
CCL2-mediated endothelial injury drives cardiac dysfunction in long COVID
Endothelial dysfunction in ME/CFS patients
Cluster Analysis to Identify Long COVID Phenotypes Using 129Xe Magnetic Resonance Imaging: A Multi-centre Evaluation
The immunology of long COVID
Sex differences and immune correlates of Long Covid development, symptom persistence, and resolution
Long COVID manifests with T cell dysregulation, inflammation and an uncoordinated adaptive immune response to SARS-CoV-2
Tissue-based T cell activation and viral RNA persist for up to 2 years after SARS-CoV-2 infection
Immunometabolic changes and potential biomarkers in CFS peripheral immune cells revealed by single-cell RNA sequencing
Distinguishing features of Long COVID identified through immune profiling
Divergent adaptive immune responses define two types of long COVID
Stress-Induced Transcriptomic Changes in Females with Myalgic Encephalomyelitis/Chronic Fatigue Syndrome Reveal Disrupted Immune Signatures
SARS-CoV-2 reservoir in post-acute sequelae of COVID-19 PASC
Association of vascular netosis with COVID-19 severity in asymptomatic and symptomatic patients
Epigenetic memory of coronavirus infection in innate immune cells and their progenitors
Transcriptional reprogramming from innate immune functions to a pro-thrombotic signature by monocytes in COVID-19
WASF3 disrupts mitochondrial respiration and may mediate exercise intolerance in myalgic encephalomyelitis/chronic fatigue syndrome
Urine Metabolomics Exposes Anomalous Recovery after Maximal Exertion in Female ME/CFS Patients
Plasma metabolomics reveals disrupted response and recovery following maximal exercise in myalgic encephalomyelitis/chronic fatigue syndrome
Altered Fatty Acid Oxidation in Lymphocyte Populations of Myalgic Encephalomyelitis/Chronic Fatigue Syndrome
A map of metabolic phenotypes in patients with myalgic encephalomyelitis/chronic fatigue syndrome
An Isolated Complex V Inefficiency and Dysregulated Mitochondrial Function in Immortalized Lymphocytes from ME/CFS Patients
SARS-CoV-2 Mitochondrial Metabolic and Epigenomic Reprogramming in COVID-19
Core mitochondrial genes are down-regulated during SARS-CoV-2 infection of rodent and human hosts
Fibrin drives thromboinflammation and neuropathology in COVID-19
Dysregulated platelet function in patients with postacute sequelae of COVID-19
Analysis of thrombogenicity under flow reveals new insights into the prothrombotic state of patients with post-COVID syndrome
Data-independent LC-MS/MS analysis of ME/CFS plasma reveals a dysregulated coagulation system, endothelial dysfunction, downregulation of complement machinery
Persistent clotting protein pathology in Long COVID/Post-Acute Sequelae of COVID-19 PASC is accompanied by increased levels of antiplasmin
Cell invasive amyloid assemblies from SARS-CoV-2 peptides can form multiple polymorphs with varying neurotoxicity
Neurotoxic amyloidogenic peptides in the proteome of SARS-COV2: potential implications for neurological symptoms in COVID-19
Proteomic Evidence for Amyloidogenic Cross-Seeding in Fibrinaloid Microclots
Amyloidogenesis of SARS-CoV-2 Spike Protein
Management of Nutritional Failure in People with Severe ME/CFS: Review of the Case for Supplementing NICE Guideline NG206
Life-Threatening Malnutrition in Very Severe ME/CFS
Maeve Boothby O'Neill: Prevention of Future Deaths Report
Caring for the Patient with Severe or Very Severe Myalgic Encephalomyelitis/Chronic Fatigue Syndrome
Graded exercise therapy for myalgic encephalomyelitis/chronic fatigue syndrome is not effective and unsafe. Re-analysis of a Cochrane review
Rethinking the treatment of chronic fatigue syndrome-a reanalysis and evaluation of findings from a recent major trial of graded exercise and CBT
The biological challenge of myalgic encephalomyelitis/chronic fatigue syndrome: a solvable problem
Last updated 17/11/24